NEW YORK (Reuters Health) Aug 13 – The molecule fetuin may play a significant role in the regulation of postprandial glucose disposal, insulin sensitivity, weight gain and fat
accumulation, researchers at Wayne State University report in the August issue of Diabetes.
Fetuin also “may be a novel therapeutic target in the treatment of type 2 diabetes, obesity,
and other insulin-resistant conditions,” they write. Dr. Suresh T. Mathews and colleagues discovered that knockout mice, which carry two null alleles for the gene that encodes fetuin, “demonstrate increased basal and insulin-stimulated phosphorylation of insulin receptor” as well as enhanced glucose clearance and enhanced insulin sensitivity.
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