Contrary to previous findings in animal models, results of a new study indicate that natural killer T lymphocytes (NKT) defects do not appear to be involved in the pathogenesis of type 1 diabetes.
Dr. Albert Bendelac from the University of Chicago, and colleagues note that earlier research has suggested that NKT cells might have a general role in suppressing autoimmune reactions. While it has been inferred from mouse models of insulin-dependent diabetes that defects in NKT cells are involved, the function and identity of NKT cells was established only through indirect methods, the researchers note. Dr. Bendelac’s team has now developed a direct method,
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